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One discussion that emerged after the talk was the question of what the right level of analysis was for understanding brain dysfunction in aging and PD, and what form of causal argument or mechanism can be derived from these network descriptions of brain organization and dysfunction. A very interesting direction to go would be to create more theoretically driven models of brain dysfunction in PD, that might explain the disconnect between the functional network effects and known pathology in the disease. These models could then be tested in future experiments.
Here are some references to our work that I discussed which could be relevant: